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General Pathology Quiz

1. Pathologies of domestic animals can be summarized in 100 words. 2. Individual cells may suffer injury through mechanisms like fatty change, necrosis, or apoptosis. 3. The main mechanisms leading to fatty change are increased fatty acid uptake and impaired fatty acid oxidation.

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Matt Dicko
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468 views

General Pathology Quiz

1. Pathologies of domestic animals can be summarized in 100 words. 2. Individual cells may suffer injury through mechanisms like fatty change, necrosis, or apoptosis. 3. The main mechanisms leading to fatty change are increased fatty acid uptake and impaired fatty acid oxidation.

Uploaded by

Matt Dicko
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as RTF, PDF, TXT or read online on Scribd
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Genpath quiz - it's really general !!

1) Summarise all pathologies of domestic animals in 100 words


2) escri!e the main ways in which an indi"idual cell may suffer in#ury
$) escri!e the $ main mechanisms leading to fatty change
%) &rue or false fatty change is irre"ersi!le
') escri!e the type of inflammatory infiltrate following autolysis
() escri!e the microscopic appearance of a necrotic cell
)) *hich type of necrosis tends to preser"e general tissue architecture+
,) -nremo"ed necrotic de!ris in a li"ing animal tends to attract which mineral+
.) /ist $ possi!le aetiologies leading to apoptosis of a cell
10) escri!e the difference !etween an e0udate1 transudate and pus2
11) escri!e the three main "ascular changes in inflammation and their causes
12) escri!e $ important outcomes of the "ascular changes in inflammation
1$) *hat acti"ates endothelial cells and white !lood cells respecti"ely in the cellular phase of
inflammation+
1%) 3ame two prominent chemotactic molecules in inflammation
1') 3ame an adhesion molecule e0pressed on endothelial cells as a result of acti"ation
1() &rue or false1 destruction of a phagolysosome's contents after phagocytosis always requires
the presence of o0ygen
1)) 4hedia5-6igashi syndrome is a defect in what+ *hich !reed of cat is predisposed+ 7s the
disease common or rare+
1,) &he most important chemical mediators of inflammation in the complement family are8 4'a1
4$a1 4$!1 and 9:4s2 escri!e what each of them do in respect to mediating inflammation2
1.) *hich of the following are plasma-deri"ed mediators and which are released from cells
;cell-deri"ed)8
4omplement system
<inin system ;!rady5inin)
=rostaglandins
/eu5otrienes
3itric o0ide
>i!rinolytic system ;plasmin)
6istamine
4oagulation system ;throm!in and friends)
20) *hat are the main effects of =G721 and what is the common name for this molecule+
21) :t low concentrations1 =:> has pretty much the same effects as which other common
chemical mediator of inflammation+ *hat are those effects+
22) 3ame an e0ogenous and an endogenous pyrogen2 6ow do they cause pyre0ia in the acute
phase response+
2$) 7n the acute phase response do serum copper le"els tend to increase or decrease+
2%) &rue or false1 serous inflammation tends to !e an e0udate
2') *hat is the difference !etween ulceration and erosion+
2() *hy is fi!rinous inflammation usually 5nown as a se"ere inflammatory change compared to
say1 serous inflammation+
2)) Grossly what does chronic inflammation tend to loo5?feel li5e+
2,) ifferentiate granulomatous and granuloma and granulation tissue2
2.) *hat are the main three white !lood cells seen in chronic inflammation+

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